diff --git a/Sweet-Relief-Glycogen-Support-Reviews-%2AIS-Legit%2A-2025-Updated-Report%21.md b/Sweet-Relief-Glycogen-Support-Reviews-%2AIS-Legit%2A-2025-Updated-Report%21.md new file mode 100644 index 0000000..8b3a3e6 --- /dev/null +++ b/Sweet-Relief-Glycogen-Support-Reviews-%2AIS-Legit%2A-2025-Updated-Report%21.md @@ -0,0 +1 @@ +
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Figure 5. The mix of recording the stimulus evoked CAP (triangle) with real time recordings of lactate (black line) allow for a fuller image of the cellular interactions to emerge. At the onset of aglycemia the lactate falls virtually instantly followed by the CAP. [Glucose](https://www.paramuspost.com/search.php?query=Glucose&type=all&mode=search&results=25) is taken up by astrocytes and either stored as glycogen or instantly processed glycolytically to lactate. The lactate is then transported to the axons for oxidative metabolism. Removing glucose from the aCSF or inhibiting glycogen metabolism with DAB or isofagomine causes the lactate to rapidly fall to zero followed by the CAP. The conclusion from this optic nerve information is that astrocytes contain glycogen and launch glycogen derived lactate into the interstitial area by way of the MCT1, from the place it is taken up into axons through the MCT2 transporter. Along with this lactate release below situations of aglycemia, where the latency to CAP failure will be accelerated by blocking lactate uptake or inhibiting glycogen metabolism, lactate can be launched tonically beneath baseline circumstances.

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